WEIGHT LOSS ……DIETS DON’T WORK. MCTS DO.

While you may have lost some weight dieting, were you able to keep it off for good? If not, you’re in the majority. This is because diets are fundamentally unsustainable.

To lose excess body weight healthfully and permanently, it takes patience and quality upgrades to your eating and exercise habits.

The good news? Lasting weight loss IS attainable when you:

  • Dedicate yourself to a nutrient-dense diet
  • Increase your activity level
  • Let go of counting calories and focus on creating healthy habits that last

We understand this is a slow progression and tough to stick to, which is why we created our MCT products to help expedite the process and promote quick weight loss.

FIGHT FAT WITH FAT

The idea that eating healthful fat makes you fat is a myth. Eating refined carbohydrates, sugar, trans fat, and highly processed vegetable oils makes you fat!

The body is an adaptive organism regulating hormone secretion and enzyme production in response to the food we eat.

So, when you ingest more MCTs, your body becomes more efficient at mobilizing fat stores as energy.

As a result, even in the presence of some carbohydrates, the body will begin breaking down its own fat to power ordinary, everyday functions. (Which means quick weight loss!)

Here are some compelling studies that show how incorporating MCT oil can help you lose the weight and keep it off:

  • MCTs given over a 6-day period was shown to boost metabolism by 50%
  • Metabolism may remain elevated for at least 24 hours after eating a meal that includes MCTs
  • During a high-calorie diet consisting of either 40% fat as MCTs or 40% fat as LCTs, the MCT group burned almost 2x the number of calories in the LCT group
  • About 1-2 TBSP of MCT oil per day resulted in a lower endpoint body weight than did the same amount of olive oil during a 16-week weight-loss program. Also, those in the MCT oil group trended towards greater fat loss and less abdominal fat mass than those in the olive oil group
  • After eating a meal containing MCTs, normal-weight individuals increased their energy expenditure by as much as 48%, while overweight participants increased theirs by as much as 65%

Several books, papers, and studies support the claims that MCT can aid in weight loss, increase physical endurance, help maintain brain health, and provide a line of defense against disease.

As new MCT research is released, it will be added to this list.

Baba, N. (1982).
Enhanced thermogenesis and diminished deposition of fat in response to overfeeding with diet containing medium-chain triglyceride.American Journal of Clinical Nutrition, 35.

Behrend, A.M, Harding, C. O., Shoemaker, J. D., Matern, D., Sahn, D. J., Elliot, D. L. & Gillingham, M. B. (2012).
Substrate oxidation and cardiac performance during exercise in disorders of long chain fatty acid oxidation. Molecular Genetics and Metabolism, 105(1).

Berry, E. M. (1997).
Dietary fatty acids in the management of diabetes mellitus. American Journal of Clinical Nutrition, 66.

Bray, G. A., Cee, M. & Bray, T. L. (1980).
Weight gain of rats fed medium-chain triglycerides is less than rats fed long-chain triglycerides. International Journal of Obesity, 4.

Clegg, M. E., Golsorkhi, M. & Henry, C. J. (2012).
Combined medium-chain triglyceride and chili feeding increase diet-induced thermogenesis in normal-weight humans. European Journal of Nutrition.

Cohen, L. A. (1988).
Medium chain triglycerides lack tumor-promoting effects in the n-methylnitrosourea-induced mammary tumor model. In The Pharmacological Effects of Lipids, vol. 3, edited by J. J. Kabara. Champaign, Illinois: The American Oil Chemists’ Society.

Cohen, L. A. & Thompson, D. O. (1987).
The influence of dietary medium chain triglycerides on rat mammary tumor development. Lipidsm 22(6).

Constantini, L. C., Barr, L. J., Vogel, J. L. & Hendersen, S. T. (2008).
Hypometabolism as a therapeutic target in Alzheimer’s disease. BMC Neuroscience, 9.

Divi, R. L., Chang, H. C. & Doerge, D. R. (1997).
Anti-thyroid isoflavones from soybean: Isolation, characterization, and mechanisms of action.  Biochemical Pharmacology, 54(10).

Duan, W., Guo, Z., Ware, M., Li, X. J. & Mattson, M. P. (2003). 
Dietary restriction normalizes glucose metabolism and BDNF levels, slows disease progression, and increases survival in huntingtin mutant mice. Proceedings of National Academy of Sciences USA, 100(5).

Enig, M. G. (1999).
Coconut: In support of good health in the twenty-first century. Paper presented at the Thirty-Sixth Annual Meeting of the APCC.

Enig, M. G. (2000).
Know your fats: The complete primer for understanding the nutrition of fats, oils, and cholesterol. Silver Spring, Maryland: Bethesda Press.

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Coconut Ketones: Fueling Brain Function & Reversing Autism. Well Being Journal, 21(5).

Fife, B. (2012).
Conquering Alzheimer’s with Coconut Ketones. Coconut Research Center.

Fife, B. (2004).
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Fine, E. J., Miller, A., Quadros, E. V., Sequeria, J. M. & Feinman, R. D. (2009).
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Fushiki, T., Matsumoto, K., Inoue, K., Kawada, T. & Sugimoto, E. (1995).
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Overfeeding with medium-chain triglycerides diet results in diminished deposition of fat. American Journal of Clinical Nutrition, 37.

Geliebter, A., Torbay, N., Bracco, E. F., Hashim, S. A. & Van Itallie, T. B. (1983).
Overfeeding with medium-chain triglycerides diet results in diminished deposition of fat. American Journal of Clinical Nutrition, 37.

Greenberger, N. J. & Skillman, T. G. (1969).
Medium-chain triglycerides: physiologic considerations and clinical applications. New England Journal of Medicine, 280.

Hashim, S. A. & Tantibhedyangkul, P. (1987).
Medium chain triglyceride in early life: Effects on growth of adipose tissue. Lipids, 22.

Hernell, O., Ward, H., Blackberg, L. & Pereira, M. E. (1986).
Killing of Giardia lamblia by human milk lipases: An effect mediated by lipolysis of milk lipids. Journal of Infectious Diseases, 153.

Hill, J. O., Peters, J. C., Yang, D., Sharp, T., Kaler, M., Abumrad, N. N. & Greene, H. L. (1989).
Thermogenesis in humans during overfeeding with medium chain triglycerides. Metabolism, 38.

Ingle, D. L. (1999).
Dietary energy value of medium chain triglycerides. Journal of Food Science, 64(6).

Issacs, C. E., Kim, K. S. & Thomar, H. (1994).
Inactivation of enveloped viruses in human bodily fluids by purified lipid. Annals of the New York Academy of Science, 724.

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The role of milk-derived antimicrobial lipids as antiviral and antibacterial agents. In Immunology of milk and the neonate, edited by J. Mestecky, Blair C. & Ogra P. L. New York: Plenum Press.

Jiang, Z. M., Zhang, S. Y. & Wang, X. R. (1993).
A comparison of medium-chain and long-chain triglycerides in surgical patients. Annals of Surgery, 217(2).

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Fatty acids and derivatives as antimicrobial agents. In The pharmacology effect of lipids, edited by J. J. Kabara. Champaign, Illinois: American Oil Chemists’ Society.

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Antimicrobial agents derived from fatty acids. Journal of the American Oil Chemists, 61.

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Kaunitz, H. & Dayri, C. S. (1992).
Coconut oil consumption and coronary heart disease. Philippine Journal.

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Kono, H., Enomoto, N., Connor, H. D., Wheeler, M. D., Bradford, B. U., Rivera, C. A., Kadiiska, M. B., Mason, R. P. & Thurman, R. G. (2000).
Medium-chain triglycerides inhibit free radical formation and TNF-alpha production in rats given enternal ethanol. American Journal of Physiology, Gastrointestinal and Liver Physiology, 278(3).

Krotkiewski, M. (2001).
Value of VLCD supplementation with medium chain triglycerides.International Journal of Obesity and Related Metabolic Disorders, 25(9).

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Physiological effects of medium-chain triglycerides: potential agents in the prevention of obesity. Journal of Nutrition, 132(3).

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Medium-chain triglycerides increase energy expenditure and decrease adiposity in overweight men. Obesity Research, 11(3).

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